The author received an honorable PhD in mathematics and majored in engineering at MIT. He attended different universities over 17 years and studied seven academic disciplines.
He has spent ~30,000 hours in endocrinology research with an emphasis in diabetes. First, he studied six metabolic diseases and food nutrition from 2010 to 2013, then conducted his own diabetes research from 2014 to 2019. His approach is " quantitative and precision medicine " based on mathematics, physics, optical and electronics physics, engineering modeling, wave theory, energy theory, signal processing, computer science, big data analytics, statistics, machine learning, and artificial intelligence. His main focus is on preventive medicine using prediction tools. He believes that the better the prediction, the more control you have. Thus far, he has written, published, and presented more than 200 medical papers.
Title of Talk :
1.Guesstimate probable partial self-recovery of pancreatic beta cells using calculations of annualized glucose data using GH-Method: math-physical medicine
2.Risk probability of having a cardiovascular disease, stroke, or renal complications using annual segmented data of glucose and metabolism index (GH Method: math-physical medicine)
1.In this paper, the author describes his hypothesis on the probable partial self-recovery of some insulin regeneration capability of pancreatic beta cells on a type 2 diabetes (T2D) patient via his collected data of both postprandial plasma glucose (PPG) and fasting plasma glucose (FPG) during the period of 1/1/2014 to 11/23/2019. The authors research methodology is 'math-physical medicine', not 'biochemical medicine' as used in the above article. Math-physical medicine has three key steps of research methodology. He starts with observing some prominent physical phenomena from his collected biomedical big data. He then forms a reasonable hypothesis from his specific observations. Finally, he derives a few mathematical equations, if possible, to verify his hypothesis. Once verified, he can then use the same equations or formulas to reproduce (or predict) the results.
The author observed improvement in his diabetes conditions after following a stringent lifestyle management since 2014. From examining his own glucose data in 2018 including the existing vulnerable conditions of his 'damaged' beta cells due to his high carbs/sugar intake, he hypothesized that beta cells are still able to 'repair' themselves to a certain degree. This 'dual-phenomena' can be observed with his higher open and close PPG values and his ultra-high PPG values when he violated his own strict control rules of diet and exercise during the same period of pancreatic beta cells partial recovery
2.In this paper, the author describes the quantitative relationship between his risk probability of having a cardiovascular disease (CVD), stroke, or renal complications and his annual segmented data of both average daily glucose and daily metabolism index (MI) by using GH-Method: math-physical medicine. In 2014, the author applied topology concept, finite-element engineering technique, and nonlinear algebra operations to develop a mathematical metabolism model, which contains ten categories including four output categories (weight, glucose, BP, other lab-tested data including lipids & ACR) and six input categories (food, water drinking, exercise, sleep, stress, routine life patterns and safety measures). These 10 metabolic categories include approximately 500 detailed elements. He further defined a new parameter referred to as the metabolism index (MI) that has a combined score of the above metabolic categories and elements. Since 2012, he has collected and stored ~2 million data from his own body health conditions and personal lifestyle details. With this mathematical risk assessment model, he can obtain three separate risk probability percentages to offer a range of the risk prediction of having CVD, stroke, or renal complications resulting from metabolic disorders, unhealthy lifestyles, and their combined impact on the human body. This paper has demonstrated the strong effect of metabolism (including glucose) on CVD/stroke risk probability % by using the annually segmented MI (and glucose) data. It has also proven the solid influence of glucose on renal complications risk probability % using annually segmented glucose (and MI) data.
(MAJ Dr. Sorush Niknamian PhD CRFM PE
APA_FPD OMS-IV AICP MWSES MLPI MACRO MTMA MRSB FIMS FChE
EMBS FNNECOS FTSCO ANCC, AANP, ACPE, ACHE, ADA CERP APA
FMDCSCO FSASS) Ph.D. in Cell and Molecular Biology and Oncology and
Professor of Cell and Molecular Biology, Oncology and Biological Attacks. He is a
Professional Biologist, Senior Research Scientist, Chartered Scientist and
Registered His Science Council ID is: A-0008833. He is one of United Kingdom
and Indiana Cancer Consortium (ICC) certified. He is a Registered Researcher in the
U.S. Government's System for Award Management, Registered Scientist at National
Center for Biotechnology Information. He is studying Medicine/Military Medicine
at Liberty University (LU), Lynchburg, VA, United States of America. He holds
Bachelors Degree in Mechatronics Engineering, applied Mathematics, Civil
Engineering and Management. He is a Member of US Government National Science
Foundation, Member of Hera Swiss Volunteers (ID: 198036), Member of US Army
American College of Physicians Chapter, Ambassador of American Cancer Society
(ACS CAN) and former Ambassador of European Association for Cancer Research
(EACR), Active Member of Federal Health Professionals at US Army Forces and
Certified Member of American Red Cross and Food Regulatory Affairs
Professionals. He is an Honorary Board Member and contributor of Weston A Price2
Foundation (WAPF) in Washington DC, United States of America. He Holds
Honorary Doctorate of Cancer Biology and Doctor of Science from Violet Cancer
Institute.He has been written
more than 30 books in different area of science specially Medicine, Evolutionary
Biology, Nutrition and Oncology. He has published more than 120 research articles
in International and National scientific journals. He is an activist in ASCO Action
Network in Virginia, ACS CAN, ACC and FPA and Improving Heart Failure
Outcomes Through Personalized Education Improving HOPE.His research interest includes Biological Sciences, General Biology, Biomedical Sciences, Clinical Oncology,
Military Medicine, Infectious Diseases, Internal Medicine, Medical Sciences.
Title of Talk :
Benefits of Nanotechnology for Cancer
Nanoscale devices are one hundred to ten thousand times smaller than human cells. They are similar in size to large biological molecules ("biomolecules") such as enzymes and receptors. As an example, hemoglobin, the molecule that carries oxygen in red blood cells, is approximately 5 nanometers in diameter. Nanoscale devices smaller than 50 nanometers can easily enter most cells, while those smaller than 20 nanometers can move out of blood vessels as they circulate through the body. Because of their small size, nanoscale devices can readily interact with biomolecules on both the surface and inside cells. By gaining access to so many areas of the body, they have the potential to detect disease and deliver treatment in ways unimagined before now. Biological processes, including ones necessary for life and those that lead to cancer, occur at the nanoscale. Thus, in fact, we are composed of a multitude of biological nano-machines. Nanotechnology provides researchers with the opportunity to study and manipulate macromolecules in real time and during the earliest stages of cancer progression. Nanotechnology can provide rapid and sensitive detection of cancer-related molecules, enabling scientists to detect molecular changes even when they occur only in a small percentage of cells. Nanotechnology also has the potential to generate entirely novel and highly effective therapeutic agents. Ultimately and uniquely, the use of nanoscale materials for cancer, comes down to its ability to be readily functionalized and easily tuned; its ability to deliver and / or act as the therapeutic, diagnostic, or both; and its ability to passively accumulate at the tumor site, to be actively targeted to cancer cells, and to be delivered across traditional biological barriers in the body such as dense stromal tissue of the pancreas or the blood-brain barrier that highly regulates delivery of biomolecules to / from, our central nervous system.
Dr.Jaleel Kareem Ahmed has completed his PhD from Baghdad University. He is the Dean of the Institute of Foundry and Hammering. He has registered 8 patents with 40 published papers and 3 books. He is a member in Who is Who network. He is a reviewer in Jon Wily and Sons and Editorial Board Member of Science Publishing Group and a member in Encyclopedia of Chemistry Scientists. He has got the Iraqi Scientist Medal. Currently, he is a Professor of physical chemistry in the College of Materials Engineering , Babylon University, Iraq.
Title of Talk :
Chlorophyll Medical Cream
In this patent a chlorophyll medical cream is prepared with high activity and healthy to protect all types of skins. The cream treats micro wound, skin roughness and clearance in addition to protect skin from sun burning cancer due to the activity of chlorophyll as a good absorbent for visible, ultra violet, x-ray and gamma ray and sun light and dissipated rays to harmless heat. Chlorophyll also heals wounds due to lowering the growth rate of harm bacteria. The cream also contain in addition to the chlorophyll other materials benefits to the skin such as Bees Wax which is used in most of the cosmetic material and for skin scare and gives a good chance to the skin to be healthy and safety respiration processes and acts as skin humidifier and inhabits babies from skin rash. Also cream contain olives oil and iodine. Iodine sublimes at room temperature from the solid crystal to vapor molecule without passing through liquid state with a radius of the molecule 0.127 nanometer, thus it's function as Nano particles sterilize and seal porous points and stop bleeding.
All materials in the cream verify their percentages according to the weather and type of the work of the users.
Creams contain chlorophyll, bees wax, olive oil and iodine which are food materials and test show all of them absorbed visible-ultraviolet (sunlight) and dissipated as harmless heat.
A research published in USA by us entitled " chlorophyll acts as gamma absorbent to protect Iraqi children from cancer" by using green milk with good result obtained.
Dr.Mahmoud Ahmed Mansour is professor at King Saud bin Abdulaziz University, Saudi Arabia.His research interests are Leukotrienes: Synthesis, Quantitation and Biological effect,Tumor Markers in the early diagnosis of cancer and Molecular diagnosis of the disease.The current reseach work interest is the exploring the role of proteasome inhibitor (reversible and irreversible) in the process of carcinogenesis and as treatment of the hepatic cancer. I will investigate the role proteasome inhibitor as a prophylactic and treatment using transgenic animal model. Moreover the cancer cell line model will also implemented. prof.Mahmoud Ahmed Mansour is the editorial member of International Journal of Cancer Immunology and Immunotherapy.
Title of Talk :
Possible role of selective, irreversible, proteasome inhibitor (carfilzomib) in the treatment of rat hepatocellular carcinoma
We investigated the possible therapeutic effect of irreversible proteasome inhibitor, carfilzomib against hepatocellular carcinoma.
Hepatocellular carcinoma induced chemically by chronic administration of diethylnitrosoamines (DENA).
Hepatocellular carcinoma induced by DENA in male Wistar rats was manifested biochemically by significant elevation of serum a-feto protein (AFP) and carcinoembryonic antigen (CEA). In addition, hepatic cancer was further confirmed by a significant increase in hepatic tissue growth factors; vascular endothelial growth factor (VEGF), transforming growth factor-b1 (TGF-b1) and basic fibroblast growth factor (FGF). Moreover a marked increase in matrix metalloproteinase-2 (MMP-2) and tissue inhibitor of metalloproteinase-1 (TIMP-1) content were also observed, along with a profound decrease in hepatic endostatin and metallothionein level.
Treatment of rats with the selected doses of carfilzomib produced a significant protection against hepatic cancer. The present results claimed that chosen doses of carfilzomib succeeded in suppressing serum tumor markers level AFP and CEA. Furthermore, the drug reduced the elevated level of hepatic growth factors, MMP-2 and TIMP-1 induced by the carcinogen. The antitumor effect of carfilzomib was also accompanied by augmentation of hepatic content of endostatin and metallothionein. Histopathological examination of liver tissues also correlated with the biochemical observations.
It could be concluded that treatment with carfilzomib confers a possible antitumor effect against hepatocellular carcinoma induced by DENA model in rats.
Dr.Cheng-Chen Chang has completed his MD from Yang-Ming Medical University and PhD from Chungshan Medical University, Institute of Medicine, Taiwan. He is the director of Department of Psychiatryand the chief of neuropsychiatric labat Changhua Christian Hospital. He has published more than 20 papers in reputed journals.
Title of Talk :
No alteration of mitochondrial DNA copy number and cytokines after short-term antipsychotic treatment in acute schizophrenic patients
Evidence suggests that mitochondrial dysfunction and cytokines are involved in the pathophysiology of schizophrenia (SZ)(Karry, Klein, & Ben Shachar, 2004; Maurer & Moller, 1997; Momtazmanesh, Zare-Shahabadi, & Rezaei, 2019)However, the exact mechanisms underlying these dysfunctions are not well understood. On the other way, mitochondrial electron transport chain complexes (ETC), and mitochondrial complex respiratory rate were inhibited by some typical and atypical antipsychotics in in-vitro and cell-line studies (Cikankova, Fisar, Bakhouche, Luptak, & Hroudova, 2019; Elmorsy & Smith, 2015). In addition, mitochondrial DNA copy number (MCN) isinversely associated with psychosis severityand antipsychotic treatment in a cross-sectional study (Kumar et al., 2018). Genome-wide association studies supportthe genetic aspects of the connection betweenschizophrenia and immune system(Stefansson et al., 2009) and the immune response is mainly mediated by cytokines.In terms of antipsychotic effect on MCN and cytokines, limited work has assessed these issues in a prospective longitudinal study. We aim to investigate the alteration of MCN and cytokines after 4-week antipsychotic treatment in acute schizophrenic patients.21 patients met DSM-5 diagnostic criteria for schizophreniawere recruited from the psychiatric inpatient unit at Changhua Christian Hospital, Taiwan from January to December in 2016. These patients are drug-naÃ¯ve or have discontinued antipsychotic drugs for at least one month. After 4-week antipsychotic treatment, we found no alteration of MCNbut IL-6 was significantly decreased despite the psychotic symptoms improved. Further exploration is needed.